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🧬 🐁 A full understanding of the functional consequences of genomic variation is crucial to using the mouse as a model for human disease. New in @cellpress Genomics, JAX's Beck lab resolves genome-wide structural variants in 20 genetically distinct inbred mice, substantially improving our understanding of transposable element contributions. The authors provide a sequence-resolved structural variant resource, a mouse embryonic stem cell expression resource, and chromatin accessibility data and s
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